Effects of HIV-1 gp120 and TAT-derived microvesicles on endothelial cell function

The aims of this study were twofold. The first was to determine if human immunodeficiency virus (HIV)-1 glycoprotein (gp) 120 and transactivator of transcription (Tat) stimulate the release of endothelial microvesicles (EMVs). The second was to determine whether viral protein-induced EMVs are delete...

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Pubblicato in:J Appl Physiol (1985)
Autori principali: Hijmans, Jamie G., Stockelman, Kelly, Levy, Ma’ayan, Brewster, L. Madden, Bammert, Tyler D., Greiner, Jared J., Connick, Elizabeth, DeSouza, Christopher A.
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Physiological Society 2019
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6589811/
https://ncbi.nlm.nih.gov/pubmed/30789287
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.01048.2018
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