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Mouse Model of Hyperkinesis Implicates SNAP-25 in Behavioral Regulation
Although hyperkinesis is expressed in several neurological disorders, the biological basis of this phenotype is unknown. The mouse mutant coloboma (Cm/+) exhibits profound spontaneous locomotor hyperactivity resulting from a deletion mutation. This deletion encompasses several genes including Snap,...
Kaydedildi:
| Yayımlandı: | J Neurosci |
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| Asıl Yazarlar: | , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Society for Neuroscience
1996
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6579059/ https://ncbi.nlm.nih.gov/pubmed/8622140 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.16-09-03104.1996 |
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