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Mouse Model of Hyperkinesis Implicates SNAP-25 in Behavioral Regulation

Although hyperkinesis is expressed in several neurological disorders, the biological basis of this phenotype is unknown. The mouse mutant coloboma (Cm/+) exhibits profound spontaneous locomotor hyperactivity resulting from a deletion mutation. This deletion encompasses several genes including Snap,...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Hess, Ellen J., Collins, Katherine A., Wilson, Michael C.
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 1996
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6579059/
https://ncbi.nlm.nih.gov/pubmed/8622140
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.16-09-03104.1996
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