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Mitochondrial Depolarization in Glutamate-Stimulated Neurons: An Early Signal Specific to Excitotoxin Exposure

A brief exposure to high concentrations of glutamate kills cultured forebrain neurons by an excitotoxic process that is dependent on Ca(2+) influx through the NMDA receptor. In this study, we have measured striking changes in mitochondrial function during and immediately after intense glutamate rece...

Ausführliche Beschreibung

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Bibliographische Detailangaben
Veröffentlicht in:J Neurosci
Hauptverfasser: White, R. James, Reynolds, Ian J.
Format: Artigo
Sprache:Inglês
Veröffentlicht: Society for Neuroscience 1996
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6578963/
https://ncbi.nlm.nih.gov/pubmed/8795624
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.16-18-05688.1996
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