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Mitochondrial Depolarization in Glutamate-Stimulated Neurons: An Early Signal Specific to Excitotoxin Exposure
A brief exposure to high concentrations of glutamate kills cultured forebrain neurons by an excitotoxic process that is dependent on Ca(2+) influx through the NMDA receptor. In this study, we have measured striking changes in mitochondrial function during and immediately after intense glutamate rece...
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| Vydáno v: | J Neurosci |
|---|---|
| Hlavní autoři: | , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Society for Neuroscience
1996
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6578963/ https://ncbi.nlm.nih.gov/pubmed/8795624 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.16-18-05688.1996 |
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