Evidence that glycine mediates the postsynaptic potentials that inhibit lumbar motoneurons during the atonia of active sleep

Lignende værker

• The postsynaptic inhibitory control of lumbar motoneurons during the atonia of active sleep: effect of strychnine on motoneuron properties
  af: Soja, PJ, et al.
  Udgivet: (1991)
• Effects of excitatory amino acid antagonists on the phasic depolarizing events that occur in lumbar motoneurons during REM periods of active sleep
  af: Soja, PJ, et al.
  Udgivet: (1995)
• Postsynaptic Inhibition of Hypoglossal Motoneurons Produces Atonia of the Genioglossal Muscle During Rapid Eye Movement Sleep
  af: Fung, Simon J., et al.
  Udgivet: (2015)
• Glycine-Mediated Postsynaptic Inhibition is Responsible for REM Sleep Atonia: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
  af: Soja, Peter J.
  Udgivet: (2008)
• Confirmation of the Consensus that Glycinergic Postsynaptic Inhibition is Responsible for the Atonia of REM Sleep: Commentary on Brooks PL and Peever JH. Glycinergic and GABA(A)-mediated inhibition of somatic motoneurons does not mediate rapid eye movement sleep motor atonia. J Neurosci 2008;28:3535–45.
  af: Chase, Michael H.
  Udgivet: (2008)