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Zinc alters excitatory amino acid neurotoxicity on cortical neurons

Recent studies have suggested that large amounts of free zinc may be coreleased during excitatory synaptic transmission at glutamatergic synapses, and may act postsynaptically to decrease actions mediated by N-methyl-D-aspartate (NMDA) receptors, while often increasing neuroexcitation mediated by qu...

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Podrobná bibliografie
Vydáno v:J Neurosci
Hlavní autoři: Koh, JY, Choi, DW
Médium: Artigo
Jazyk:Inglês
Vydáno: Society for Neuroscience 1988
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6569314/
https://ncbi.nlm.nih.gov/pubmed/3385493
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.08-06-02164.1988
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