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Zinc alters excitatory amino acid neurotoxicity on cortical neurons
Recent studies have suggested that large amounts of free zinc may be coreleased during excitatory synaptic transmission at glutamatergic synapses, and may act postsynaptically to decrease actions mediated by N-methyl-D-aspartate (NMDA) receptors, while often increasing neuroexcitation mediated by qu...
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| Foilsithe in: | J Neurosci |
|---|---|
| Main Authors: | , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Society for Neuroscience
1988
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6569314/ https://ncbi.nlm.nih.gov/pubmed/3385493 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.08-06-02164.1988 |
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