MON-201 ER Stress Activated by Androgen Enhances Apoptosis of Granulosa Cells via Induction of Death Receptor 5 in PCOS

Lignende værker

• MON-033 Androgen Increases the Accumulation of Advanced Glycation End Products in Granulosa Cells by Activating ER Stress in PCOS
  af: Azhary, Jerilee Mariam Khong, et al.
  Udgivet: (2020)
• Upregulation of Aryl Hydrocarbon Receptor in Granulosa Cells by Endoplasmic Reticulum Stress Contributes to the PCOS Pathophysiology
  af: Kunitomi, Chisato, et al.
  Udgivet: (2021)
• Activation of Endoplasmic Reticulum Stress in Granulosa Cells from Patients with Polycystic Ovary Syndrome Contributes to Ovarian Fibrosis
  af: Takahashi, Nozomi, et al.
  Udgivet: (2017)
• MON-027 Activation of Endoplasmic Reticulum Stress Mediates Oxidative Stress-Induced Apoptosis of Granulosa Cells in Ovaries Affected by Endometrioma
  af: Kunitomi, Chisato, et al.
  Udgivet: (2020)
• Assisted reproductive technology pregnancy complications are significantly associated with endometriosis severity before conception: a retrospective cohort study
  af: Tatsuya Fujii, et al.
  Udgivet: (2016-11-01)