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Augmented β2-adrenergic signaling dampens the neuroinflammatory response following ischemic stroke and increases stroke size
BACKGROUND: Ischemic stroke provokes a neuroinflammatory response and simultaneously promotes release of epinephrine and norepinephrine by the sympathetic nervous system. This increased sympathetic outflow can act on β2-adrenergic receptors expressed by immune cells such as brain-resident microglia...
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| Udgivet i: | J Neuroinflammation |
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| Main Authors: | , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
BioMed Central
2019
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6540436/ https://ncbi.nlm.nih.gov/pubmed/31138227 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12974-019-1506-4 |
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