Augmented β2-adrenergic signaling dampens the neuroinflammatory response following ischemic stroke and increases stroke size

BACKGROUND: Ischemic stroke provokes a neuroinflammatory response and simultaneously promotes release of epinephrine and norepinephrine by the sympathetic nervous system. This increased sympathetic outflow can act on β2-adrenergic receptors expressed by immune cells such as brain-resident microglia...

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Detalhes bibliográficos
Publicado no:J Neuroinflammation
Main Authors: Lechtenberg, Kendra J., Meyer, Scott T., Doyle, Janelle B., Peterson, Todd C., Buckwalter, Marion S.
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2019
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6540436/
https://ncbi.nlm.nih.gov/pubmed/31138227
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12974-019-1506-4
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