Lateral hypothalamic orexin glucose-inhibited neurons may regulate reward-based feeding by modulating glutamate transmission in the ventral tegmental area

Glucose inhibits ~60% of lateral hypothalamic (LH) orexin neurons. Fasting increases the activation of LH orexin glucose-inhibited (GI) neurons in low glucose. Increases in spontaneous glutamate excitatory postsynaptic currents (sEPSCs) onto putative VTA DA neurons in low glucose are orexin dependen...

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Foilsithe in:Brain Res
Main Authors: Teegala, Suraj B., Sheng, Zhenyu, Dalal, Miloni S., Hirschberg, Pamela R., Beck, Kevin D., Routh, Vanessa H.
Formáid: Artigo
Teanga:Inglês
Foilsithe: 2018
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Article
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6525648/
https://ncbi.nlm.nih.gov/pubmed/29787770
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.brainres.2018.05.025
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