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A Tripartite Interaction Among the Calcium Channel α(1)- and β-Subunits and F-Actin Increases the Readily Releasable Pool of Vesicles and Its Recovery After Depletion
Neurotransmitter release is initiated by the influx of Ca(2+) via voltage-gated calcium channels. The accessory β-subunit (Ca(V)β) of these channels shapes synaptic transmission by associating with the pore-forming subunit (Ca(V)α(1)) and up-regulating presynaptic calcium currents. Besides Ca(V)α(1,...
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| Publicat a: | Front Cell Neurosci |
|---|---|
| Autors principals: | , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Frontiers Media S.A.
2019
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6509170/ https://ncbi.nlm.nih.gov/pubmed/31130843 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2019.00125 |
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