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A Tripartite Interaction Among the Calcium Channel α(1)- and β-Subunits and F-Actin Increases the Readily Releasable Pool of Vesicles and Its Recovery After Depletion
Neurotransmitter release is initiated by the influx of Ca(2+) via voltage-gated calcium channels. The accessory β-subunit (Ca(V)β) of these channels shapes synaptic transmission by associating with the pore-forming subunit (Ca(V)α(1)) and up-regulating presynaptic calcium currents. Besides Ca(V)α(1,...
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| Foilsithe in: | Front Cell Neurosci |
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| Main Authors: | , , , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Frontiers Media S.A.
2019
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6509170/ https://ncbi.nlm.nih.gov/pubmed/31130843 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2019.00125 |
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