Transient Lack of Glucose but not O(2) is Involved in Ischemic Postconditioning‐Induced Neuroprotection

AIM: Cerebral ischemic postconditioning has emerged recently as a kind of endogenous strategy for neuroprotection. We set out to test whether hypoxia or glucose deprivation (GD) would substitute for ischemia in postconditioning. METHODS: Adult male C57BL/6J mice were treated with postconditioning ev...

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Pubblicato in:CNS Neurosci Ther
Autori principali: Fan, Yan‐Ying, Zhang, Xiang‐Nan, He, Ping, Shen, Zhe, Shen, Yao, Wang, Xiao‐Fen, Hu, Wei‐Wei, Chen, Zhong
Natura: Artigo
Lingua:Inglês
Pubblicazione: John Wiley and Sons Inc. 2012
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Original Articles
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6493356/
https://ncbi.nlm.nih.gov/pubmed/23167958
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cns.12033
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