Transient Lack of Glucose but not O(2) is Involved in Ischemic Postconditioning‐Induced Neuroprotection

AIM: Cerebral ischemic postconditioning has emerged recently as a kind of endogenous strategy for neuroprotection. We set out to test whether hypoxia or glucose deprivation (GD) would substitute for ischemia in postconditioning. METHODS: Adult male C57BL/6J mice were treated with postconditioning ev...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Veröffentlicht in:CNS Neurosci Ther
Hauptverfasser: Fan, Yan‐Ying, Zhang, Xiang‐Nan, He, Ping, Shen, Zhe, Shen, Yao, Wang, Xiao‐Fen, Hu, Wei‐Wei, Chen, Zhong
Format: Artigo
Sprache:Inglês
Veröffentlicht: John Wiley and Sons Inc. 2012
Schlagworte:
Original Articles
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6493356/
https://ncbi.nlm.nih.gov/pubmed/23167958
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/cns.12033
Tags: Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!

Ähnliche Einträge