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Mutant p53 in cancer therapy—the barrier or the path

Since wild-type p53 is central for maintaining genomic stability and preventing oncogenesis, its coding gene TP53 is highly mutated in ~50% of human cancers, and its activity is almost abrogated in the rest of cancers. Approximately 80% of p53 mutations are single point mutations with several hotspo...

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Detalhes bibliográficos
Publicado no:J Mol Cell Biol
Main Authors: Zhou, Xiang, Hao, Qian, Lu, Hua
Formato: Artigo
Idioma:Inglês
Publicado em: Oxford University Press 2018
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6487791/
https://ncbi.nlm.nih.gov/pubmed/30508182
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/jmcb/mjy072
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