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Allicin disrupts cardiac Cav1.2 channels via trafficking
Context: Allicin is a potential antiarrhythmic agent. The antiarrhythmic properties of allicin rely on its blockade of various cardiac ion channels. The l-type calcium (Cav1.2) channel provides a pivotal substrate for cardiac electrophysiologic activities. The mechanism of allicin on Cav1.2 remains...
Salvato in:
| Pubblicato in: | Pharm Biol |
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| Autori principali: | , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Taylor & Francis
2019
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6450490/ https://ncbi.nlm.nih.gov/pubmed/30929547 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/13880209.2019.1577469 |
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