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Allicin disrupts cardiac Cav1.2 channels via trafficking
Context: Allicin is a potential antiarrhythmic agent. The antiarrhythmic properties of allicin rely on its blockade of various cardiac ion channels. The l-type calcium (Cav1.2) channel provides a pivotal substrate for cardiac electrophysiologic activities. The mechanism of allicin on Cav1.2 remains...
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| Publicat a: | Pharm Biol |
|---|---|
| Autors principals: | , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Taylor & Francis
2019
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6450490/ https://ncbi.nlm.nih.gov/pubmed/30929547 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/13880209.2019.1577469 |
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