Activation of hepatic estrogen receptor-α increases energy expenditure by stimulating the production of fibroblast growth factor 21 in female mice

OBJECTIVE: The endogenous estrogen 17β-estradiol (E2) promotes metabolic homeostasis in premenopausal women. In a mouse model of post-menopausal metabolic syndrome, we reported that estrogens increased energy expenditure, thus preventing estrogen deficiency-induced adiposity. Estrogens' prevent...

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Détails bibliographiques
Publié dans:Mol Metab
Auteurs principaux: Allard, Camille, Bonnet, Fabrice, Xu, Beibei, Coons, Laurel, Albarado, Diana, Hill, Cristal, Fagherazzi, Guy, Korach, Kenneth S., Levin, Ellis R., Lefante, John, Morrison, Christopher, Mauvais-Jarvis, Franck
Format: Artigo
Langue:Inglês
Publié: Elsevier 2019
Sujets:
Original Article
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6437689/
https://ncbi.nlm.nih.gov/pubmed/30797705
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molmet.2019.02.002
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