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Histone Methylation Directs Myeloid Toll-like Receptor 4 Expression and Regulates Wound Healing Following Cutaneous Tissue Injury
Myeloid cells, are critical for orchestrating regulated inflammation during wound healing. Toll-like receptors (TLRs), particularly TLR4 and its downstream signaling MyD88 pathway, play an important role in regulating myeloid-mediated inflammation. Since an initial inflammatory phase is vital for ti...
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| 出版年: | J Immunol |
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| 主要な著者: | , , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2019
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6401313/ https://ncbi.nlm.nih.gov/pubmed/30710046 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1801258 |
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