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RAD52 and SLX4 act nonepistatically to ensure telomere stability during alternative telomere lengthening
Approximately 15% of cancers use homologous recombination for alternative lengthening of telomeres (ALT). How the initiating genomic lesions invoke homology-directed telomere synthesis remains enigmatic. Here, we show that distinct dependencies exist for telomere synthesis in response to replication...
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| Publicado no: | Genes Dev |
|---|---|
| Main Authors: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Cold Spring Harbor Laboratory Press
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6362809/ https://ncbi.nlm.nih.gov/pubmed/30692206 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.319723.118 |
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