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Cancer-Associated Intermediate Conductance Ca(2+)-Activated K(+) Channel K(Ca)3.1

Several tumor entities have been reported to overexpress K(Ca)3.1 potassium channels due to epigenetic, transcriptional, or post-translational modifications. By modulating membrane potential, cell volume, or Ca(2+) signaling, K(Ca)3.1 has been proposed to exert pivotal oncogenic functions in tumorig...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:Cancers (Basel)
मुख्य लेखकों: Mohr, Corinna J., Steudel, Friederike A., Gross, Dominic, Ruth, Peter, Lo, Wing-Yee, Hoppe, Reiner, Schroth, Werner, Brauch, Hiltrud, Huber, Stephan M., Lukowski, Robert
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: MDPI 2019
विषय:
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC6357066/
https://ncbi.nlm.nih.gov/pubmed/30658505
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/cancers11010109
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