Contribution of systemic inflammation to permanence of K(ATP)-induced neonatal diabetes in mice

Gain-of-function (GOF) mutations in the ATP-sensitive potassium (K(ATP)) channels cause neonatal diabetes. Despite the well-established genetic root of the disease, pathways modulating disease severity and treatment effectiveness remain poorly understood. Patient phenotypes can vary from severe diab...

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Pubblicato in:Am J Physiol Endocrinol Metab
Autori principali: Emfinger, Christopher H., Yan, Zihan, Welscher, Alecia, Hung, Peter, McAllister, William, Hruz, Paul W., Nichols, Colin G., Remedi, Maria S.
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Physiological Society 2018
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6336961/
https://ncbi.nlm.nih.gov/pubmed/30226997
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpendo.00137.2018
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