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Characterisation of a type II functionally-deficient variant of alpha-1-antitrypsin discovered in the general population
Lung disease in alpha-1-antitrypsin deficiency (AATD) results from dysregulated proteolytic activity, mainly by neutrophil elastase (HNE), in the lung parenchyma. This is the result of a substantial reduction of circulating alpha-1-antitrypsin (AAT) and the presence in the plasma of inactive polymer...
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| Udgivet i: | PLoS One |
|---|---|
| Main Authors: | , , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Public Library of Science
2019
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6329500/ https://ncbi.nlm.nih.gov/pubmed/30633749 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0206955 |
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