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Adrenal tissue-specific deletion of TASK channels causes aldosterone-driven Angiotensin II-independent hypertension.
The renin-angiotensin system tightly controls aldosterone synthesis. Dysregulation is evident in hypertension (primary aldosteronism, low-renin and resistant hypertension), but also can exist in normotension. Whether chronic, mild aldosterone autonomy can elicit hypertension remains untested. Previo...
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| Yayımlandı: | Hypertension |
|---|---|
| Asıl Yazarlar: | , , , , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
2019
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6326871/ https://ncbi.nlm.nih.gov/pubmed/30580687 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/HYPERTENSIONAHA.118.11962 |
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