Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway

BACKGROUND: Neuroinflammation is closely associated with functional outcome in subarachnoid hemorrhage (SAH) patients. Our recent study demonstrated that fluoxetine inhibited NLRP3 inflammasome activation and attenuated necrotic cell death in early brain injury after SAH, while the effects and poten...

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Bibliografiska uppgifter
I publikationen:J Neuroinflammation
Huvudupphovsmän: Liu, Fu-yi, Cai, Jing, Wang, Chun, Ruan, Wu, Guan, Guo-ping, Pan, Hai-zhou, Li, Jian-ru, Qian, Cong, Chen, Jing-sen, Wang, Lin, Chen, Gao
Materialtyp: Artigo
Språk:Inglês
Publicerad: BioMed Central 2018
Ämnen:
Research
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC6302437/
https://ncbi.nlm.nih.gov/pubmed/30572907
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12974-018-1388-x
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