Fluoxetine attenuates neuroinflammation in early brain injury after subarachnoid hemorrhage: a possible role for the regulation of TLR4/MyD88/NF-κB signaling pathway

BACKGROUND: Neuroinflammation is closely associated with functional outcome in subarachnoid hemorrhage (SAH) patients. Our recent study demonstrated that fluoxetine inhibited NLRP3 inflammasome activation and attenuated necrotic cell death in early brain injury after SAH, while the effects and poten...

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Detalhes bibliográficos
Publicado no:J Neuroinflammation
Main Authors: Liu, Fu-yi, Cai, Jing, Wang, Chun, Ruan, Wu, Guan, Guo-ping, Pan, Hai-zhou, Li, Jian-ru, Qian, Cong, Chen, Jing-sen, Wang, Lin, Chen, Gao
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2018
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6302437/
https://ncbi.nlm.nih.gov/pubmed/30572907
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12974-018-1388-x
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