Rapamycin attenuates mitochondrial injury and renal tubular cell apoptosis in experimental contrast-induced acute kidney injury in rats

Reactive oxygen species (ROS) overproduction and renal tubular epithelial cell (TEC) apoptosis are key mechanisms of contrast-induced acute kidney injury (CI-AKI). Mitochondria are the main source of intracellular ROS. In the present study, the characteristics of mitophagy and the effects of rapamyc...

Celý popis

Uloženo v:
Podrobná bibliografie
Vydáno v:Biosci Rep
Hlavní autoři: Yang, Xueyan, Yan, Xiaojie, Yang, Dingping, Zhou, Junke, Song, Jie, Yang, Dingwei
Médium: Artigo
Jazyk:Inglês
Vydáno: Portland Press Ltd. 2018
Témata:
Research Articles
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6246763/
https://ncbi.nlm.nih.gov/pubmed/30341250
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/BSR20180876
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