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With no lysine kinase 4 modulates sodium potassium 2 chloride cotransporter activity in vivo

With no lysine kinase 4 (WNK4) is essential to activate the thiazide-sensitive NaCl cotransporter (NCC) along the distal convoluted tubule, an effect central to the phenotype of familial hyperkalemic hypertension. Although effects on potassium and sodium channels along the connecting and collecting...

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Detaylı Bibliyografya
Yayımlandı:Am J Physiol Renal Physiol
Asıl Yazarlar: Terker, Andrew S., Castañeda-Bueno, Maria, Ferdaus, Mohammed Z., Cornelius, Ryan J., Erspamer, Kayla J., Su, Xiao-Tong, Miller, Lauren N., McCormick, James A., Wang, Wen-Hui, Gamba, Gerardo, Yang, Chao-Ling, Ellison, David H.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Physiological Society 2018
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6230736/
https://ncbi.nlm.nih.gov/pubmed/29412704
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00485.2017
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