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With no lysine kinase 4 modulates sodium potassium 2 chloride cotransporter activity in vivo
With no lysine kinase 4 (WNK4) is essential to activate the thiazide-sensitive NaCl cotransporter (NCC) along the distal convoluted tubule, an effect central to the phenotype of familial hyperkalemic hypertension. Although effects on potassium and sodium channels along the connecting and collecting...
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| Yayımlandı: | Am J Physiol Renal Physiol |
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| Asıl Yazarlar: | , , , , , , , , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
American Physiological Society
2018
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6230736/ https://ncbi.nlm.nih.gov/pubmed/29412704 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00485.2017 |
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