With no lysine kinase 4 modulates sodium potassium 2 chloride cotransporter activity in vivo

With no lysine kinase 4 (WNK4) is essential to activate the thiazide-sensitive NaCl cotransporter (NCC) along the distal convoluted tubule, an effect central to the phenotype of familial hyperkalemic hypertension. Although effects on potassium and sodium channels along the connecting and collecting...

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Pubblicato in:Am J Physiol Renal Physiol
Autori principali: Terker, Andrew S., Castañeda-Bueno, Maria, Ferdaus, Mohammed Z., Cornelius, Ryan J., Erspamer, Kayla J., Su, Xiao-Tong, Miller, Lauren N., McCormick, James A., Wang, Wen-Hui, Gamba, Gerardo, Yang, Chao-Ling, Ellison, David H.
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Physiological Society 2018
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6230736/
https://ncbi.nlm.nih.gov/pubmed/29412704
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00485.2017
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