eNOS expression and NO release during hypoxia is inhibited by miR-200b in human endothelial cells

The nitric oxide (NO) secreted by vascular endothelium is required for the maintenance of cardiovascular homeostasis. Diminished release of NO generated by endothelial NO synthase contributes to endothelial dysfunction. Hypoxia and ischemia reduce endothelial eNOS expression via posttranscriptional...

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Kaydedildi:
Detaylı Bibliyografya
Yayımlandı:Angiogenesis
Asıl Yazarlar: Janaszak-Jasiecka, Anna, Siekierzycka, Anna, Bartoszewska, Sylwia, Serocki, Marcin, Dobrucki, Lawrence W., Collawn, James F., Kalinowski, Leszek, Bartoszewski, Rafal
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Springer Netherlands 2018
Konular:
Brief Communication
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6208887/
https://ncbi.nlm.nih.gov/pubmed/29737439
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10456-018-9620-y
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