eNOS expression and NO release during hypoxia is inhibited by miR-200b in human endothelial cells

The nitric oxide (NO) secreted by vascular endothelium is required for the maintenance of cardiovascular homeostasis. Diminished release of NO generated by endothelial NO synthase contributes to endothelial dysfunction. Hypoxia and ischemia reduce endothelial eNOS expression via posttranscriptional...

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Vydáno v:Angiogenesis
Hlavní autoři: Janaszak-Jasiecka, Anna, Siekierzycka, Anna, Bartoszewska, Sylwia, Serocki, Marcin, Dobrucki, Lawrence W., Collawn, James F., Kalinowski, Leszek, Bartoszewski, Rafal
Médium: Artigo
Jazyk:Inglês
Vydáno: Springer Netherlands 2018
Témata:
Brief Communication
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6208887/
https://ncbi.nlm.nih.gov/pubmed/29737439
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10456-018-9620-y
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