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eNOS expression and NO release during hypoxia is inhibited by miR-200b in human endothelial cells
The nitric oxide (NO) secreted by vascular endothelium is required for the maintenance of cardiovascular homeostasis. Diminished release of NO generated by endothelial NO synthase contributes to endothelial dysfunction. Hypoxia and ischemia reduce endothelial eNOS expression via posttranscriptional...
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| Veröffentlicht in: | Angiogenesis |
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| Hauptverfasser: | , , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Springer Netherlands
2018
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6208887/ https://ncbi.nlm.nih.gov/pubmed/29737439 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10456-018-9620-y |
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