eNOS expression and NO release during hypoxia is inhibited by miR-200b in human endothelial cells

The nitric oxide (NO) secreted by vascular endothelium is required for the maintenance of cardiovascular homeostasis. Diminished release of NO generated by endothelial NO synthase contributes to endothelial dysfunction. Hypoxia and ischemia reduce endothelial eNOS expression via posttranscriptional...

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Salvato in:
Dettagli Bibliografici
Pubblicato in:Angiogenesis
Autori principali: Janaszak-Jasiecka, Anna, Siekierzycka, Anna, Bartoszewska, Sylwia, Serocki, Marcin, Dobrucki, Lawrence W., Collawn, James F., Kalinowski, Leszek, Bartoszewski, Rafal
Natura: Artigo
Lingua:Inglês
Pubblicazione: Springer Netherlands 2018
Soggetti:
Brief Communication
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6208887/
https://ncbi.nlm.nih.gov/pubmed/29737439
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10456-018-9620-y
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