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Expression of calcineurin, calpastatin and heat shock proteins during ischemia and reperfusion
OBJECTIVE: Calcineurin (CaN) interacts with calpains (Calpn) and causes cellular damage eventually leading to cell death. Calpastatin (Calp) is a specific Calpn inhibitor, along with CaN stimulation has been implicated in reduced cell death and self-repair. Molecular chaperones, heat shock proteins...
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| Pubblicato in: | Biochem Biophys Rep |
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| Autori principali: | , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Elsevier
2015
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6189699/ https://ncbi.nlm.nih.gov/pubmed/30338302 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbrep.2015.09.016 |
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