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Nicotinamide mononucleotide preserves mitochondrial function and increases survival in hemorrhagic shock
Hemorrhagic shock depletes nicotinamide adenine dinucleotide (NAD) and causes metabolic derangements that, in severe cases, cannot be overcome, even after restoration of blood volume and pressure. However, current strategies to treat acute blood loss do not target cellular metabolism. We hypothesize...
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| Publicado en: | JCI Insight |
|---|---|
| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
American Society for Clinical Investigation
2018
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6171817/ https://ncbi.nlm.nih.gov/pubmed/30185676 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.120182 |
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