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Hypertrophic cardiomyopathy disease results from disparate impairments of cardiac myosin function and auto-inhibition
Hypertrophic cardiomyopathies (HCM) result from distinct single-point mutations in sarcomeric proteins that lead to muscle hypercontractility. While different models account for a pathological increase in the power output, clear understanding of the molecular basis of dysfunction in HCM is the manda...
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| Veröffentlicht in: | Nat Commun |
|---|---|
| Hauptverfasser: | , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Nature Publishing Group UK
2018
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6167380/ https://ncbi.nlm.nih.gov/pubmed/30275503 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-018-06191-4 |
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