TACI deletion protects against progressive murine lupus nephritis induced by BAFF overexpression

B cells are known to promote the pathogenesis of systemic lupus erythematosus (SLE) via the production of pathogenic anti-nuclear antibodies. However, the signals required for autoreactive B cell activation and the immune mechanisms whereby B cells impact lupus nephritis pathology remain poorly unde...

Täydet tiedot

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Bibliografiset tiedot
Julkaisussa:Kidney Int
Päätekijät: Arkatkar, Tanvi, Jacobs, Holly M., Du, Samuel W., Li, Quan-Zhen, Hudkins, Kelly L., Alpers, Charles E., Rawlings, David J., Jackson, Shaun W.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2018
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Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6151274/
https://ncbi.nlm.nih.gov/pubmed/29907458
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.kint.2018.03.012
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