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Analgesia linked to Nav1.7 loss of function requires µ- and δ-opioid receptors
Background: Functional deletion of the Scn9a (sodium voltage-gated channel alpha subunit 9) gene encoding sodium channel Nav1.7 makes humans and mice pain-free. Opioid signalling contributes to this analgesic state. We have used pharmacological and genetic approaches to identify the opioid receptors...
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| Izdano u: | Wellcome Open Res |
|---|---|
| Glavni autori: | , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
F1000 Research Limited
2018
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6134336/ https://ncbi.nlm.nih.gov/pubmed/30271888 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.12688/wellcomeopenres.14687.1 |
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