Interleukin-1β blockade improves cardiac remodeling after myocardial infarction without interrupting the inflammasome in the mouse

The formation of the cryopyrin inflammasome in the heart induces an intense inflammatory response during acute myocardial infarction (AMI), which mediates further damage and promotes adverse cardiac remodeling. Active interleukin-1β (IL-1β) is a key product of the inflammasome, being cleaved by acti...

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Detaylı Bibliyografya
Yayımlandı:Exp Physiol
Asıl Yazarlar: Toldo, Stefano, Mezzaroma, Eleonora, Van Tassell, Benjamin W., Farkas, Daniela, Marchetti, Carlo, Voelkel, Norbert F., Abbate, Antonio
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2012
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6119592/
https://ncbi.nlm.nih.gov/pubmed/23180808
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/expphysiol.2012.069831
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