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Interleukin-1β blockade improves cardiac remodeling after myocardial infarction without interrupting the inflammasome in the mouse

The formation of the cryopyrin inflammasome in the heart induces an intense inflammatory response during acute myocardial infarction (AMI), which mediates further damage and promotes adverse cardiac remodeling. Active interleukin-1β (IL-1β) is a key product of the inflammasome, being cleaved by acti...

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Détails bibliographiques
Publié dans:Exp Physiol
Auteurs principaux: Toldo, Stefano, Mezzaroma, Eleonora, Van Tassell, Benjamin W., Farkas, Daniela, Marchetti, Carlo, Voelkel, Norbert F., Abbate, Antonio
Format: Artigo
Langue:Inglês
Publié: 2012
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6119592/
https://ncbi.nlm.nih.gov/pubmed/23180808
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/expphysiol.2012.069831
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