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Interleukin-1β blockade improves cardiac remodeling after myocardial infarction without interrupting the inflammasome in the mouse
The formation of the cryopyrin inflammasome in the heart induces an intense inflammatory response during acute myocardial infarction (AMI), which mediates further damage and promotes adverse cardiac remodeling. Active interleukin-1β (IL-1β) is a key product of the inflammasome, being cleaved by acti...
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| Publié dans: | Exp Physiol |
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| Auteurs principaux: | , , , , , , |
| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
2012
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6119592/ https://ncbi.nlm.nih.gov/pubmed/23180808 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/expphysiol.2012.069831 |
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