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Mitochondrial Complex I in the Post-ischemic Heart: REPERFUSION-MEDIATED OXIDATIVE INJURY AND PROTEIN CYSTEINE SULFONATION
A serious consequence of ischemia-reperfusion injury (I/R) is oxidative damage leading to mitochondrial dysfunction. Such I/R-induced mitochondrial dysfunction is observed as impaired state 3 respiration and overproduction of (•)O(2)(−). The cascading ROS can propagate cysteine oxidation on mitochon...
Tallennettuna:
| Julkaisussa: | J Mol Cell Cardiol |
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| Päätekijät: | , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2018
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6103816/ https://ncbi.nlm.nih.gov/pubmed/30031815 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2018.07.244 |
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