Knockdown of Sestrin2 Increases Lipopolysaccharide-Induced Oxidative Stress, Apoptosis, and Fibrotic Reactions in H9c2 Cells and Heart Tissues of Mice via an AMPK-Dependent Mechanism

Sestrin2 (sesn2) is an endogenous antioxidant protein that has recently gained attention for its potential to treat various inflammatory diseases. However, the relationship of sesn2 with cardiomyopathy is still unclear. In H9c2 cells, sesn2 knockdown reduced the level of 5′ adenosine monophosphate-a...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Τόπος έκδοσης:Mediators Inflamm
Κύριοι συγγραφείς: Hwang, Hwan-Jin, Kim, Joo Won, Chung, Hye Soo, Seo, Ji A., Kim, Sin Gon, Kim, Nan Hee, Choi, Kyung Mook, Baik, Sei Hyun, Yoo, Hye Jin
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: Hindawi 2018
Θέματα:
Research Article
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6079459/
https://ncbi.nlm.nih.gov/pubmed/30116150
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2018/6209140
Ετικέτες: Προσθήκη ετικέτας
Δεν υπάρχουν, Καταχωρήστε ετικέτα πρώτοι!

Παρόμοια τεκμήρια