Knockdown of Sestrin2 Increases Lipopolysaccharide-Induced Oxidative Stress, Apoptosis, and Fibrotic Reactions in H9c2 Cells and Heart Tissues of Mice via an AMPK-Dependent Mechanism

Sestrin2 (sesn2) is an endogenous antioxidant protein that has recently gained attention for its potential to treat various inflammatory diseases. However, the relationship of sesn2 with cardiomyopathy is still unclear. In H9c2 cells, sesn2 knockdown reduced the level of 5′ adenosine monophosphate-a...

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Detalhes bibliográficos
Publicado no:Mediators Inflamm
Main Authors: Hwang, Hwan-Jin, Kim, Joo Won, Chung, Hye Soo, Seo, Ji A., Kim, Sin Gon, Kim, Nan Hee, Choi, Kyung Mook, Baik, Sei Hyun, Yoo, Hye Jin
Formato: Artigo
Idioma:Inglês
Publicado em: Hindawi 2018
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6079459/
https://ncbi.nlm.nih.gov/pubmed/30116150
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2018/6209140
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