TLR-7-mediated Lupus Nephritis Is Independent of Type I Interferon Signaling

Systemic Lupus Erythematosus (SLE) is an autoimmune disease characterized by increased type I interferons (IFNs), autoantibodies, and inflammatory-mediated multi-organ damage. TLR7 activation is an important contributor to SLE pathogenesis, but the mechanisms by which type I IFNs participate in TLR7...

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Bibliografske podrobnosti
izdano v:J Immunol
Main Authors: Wolf, Sonya J., Theros, Jonathan, Reed, Tammi J., Liu, Jianhua, Grigorova, Irina L., Martínez-Colón, Giovanny, Jacob, Chaim O., Hodgin, Jeffrey B., Kahlenberg, J. Michelle
Format: Artigo
Jezik:Inglês
Izdano: 2018
Teme:
Article
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC6039244/
https://ncbi.nlm.nih.gov/pubmed/29884703
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1701588
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