Melatonin inhibits 17β-estradiol-induced migration, invasion and epithelial-mesenchymal transition in normal and endometriotic endometrial epithelial cells

BACKGROUND: Melatonin is a potential therapeutic agent for endometriosis, but its molecular mechanism is unclear. Here, we investigated the effect of melatonin on the epithelial-mesenchymal transition (EMT) in endometriotic endometrial epithelial cells and explored the pathway that might be involved...

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Vydáno v:Reprod Biol Endocrinol
Hlavní autoři: Qi, Shasha, Yan, Lei, Liu, Zhao, Mu, Yu-lan, Li, Mingjiang, Zhao, Xingbo, Chen, Zi-Jiang, Zhang, Hui
Médium: Artigo
Jazyk:Inglês
Vydáno: BioMed Central 2018
Témata:
Research
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6015458/
https://ncbi.nlm.nih.gov/pubmed/29935526
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12958-018-0375-5
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