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Modeling the Transition From Decompensated to Pathological Hypertrophy
BACKGROUND: Long‐chain acyl‐CoA synthetases (ACSL) catalyze the conversion of long‐chain fatty acids to fatty acyl‐CoAs. Cardiac‐specific ACSL1 temporal knockout at 2 months results in a shift from FA oxidation toward glycolysis that promotes mTORC1‐mediated ventricular hypertrophy. We used unbiased...
Shranjeno v:
| izdano v: | J Am Heart Assoc |
|---|---|
| Main Authors: | , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
John Wiley and Sons Inc.
2018
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6015423/ https://ncbi.nlm.nih.gov/pubmed/29622588 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/JAHA.117.008293 |
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