Overexpression of cellular glutathione peroxidase rescues homocyst(e)ine-induced endothelial dysfunction
Homocyst(e)ine (Hcy) inhibits the expression of the antioxidant enzyme cellular glutathione peroxidase (GPx-1) in vitro and in vivo, which can lead to an increase in reactive oxygen species that inactivate NO and promote endothelial dysfunction. In this study, we tested the hypothesis that overexpre...
שמור ב:
| הוצא לאור ב: | Proc Natl Acad Sci U S A |
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| Principais autores: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
National Academy of Sciences
2001
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC60083/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11606774/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.231428998 |
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