Lipophilic components of diesel exhaust particles induce pro-inflammatory responses in human endothelial cells through AhR dependent pathway(s)

BACKGROUND: Exposure to traffic-derived particulate matter (PM), such as diesel exhaust particles (DEP), is a leading environmental cause of cardiovascular disease (CVD), and may contribute to endothelial dysfunction and development of atherosclerosis. It is still debated how DEP and other inhaled P...

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Dettagli Bibliografici
Pubblicato in:Part Fibre Toxicol
Autori principali: Brinchmann, Bendik C., Skuland, Tonje, Rambøl, Mia H., Szoke, Krisztina, Brinchmann, Jan E., Gutleb, Arno C., Moschini, Elisa, Kubátová, Alena, Kukowski, Klara, Le Ferrec, Eric, Lagadic-Gossmann, Dominique, Schwarze, Per E., Låg, Marit, Refsnes, Magne, Øvrevik, Johan, Holme, Jørn A.
Natura: Artigo
Lingua:Inglês
Pubblicazione: BioMed Central 2018
Soggetti:
Research
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5948689/
https://ncbi.nlm.nih.gov/pubmed/29751765
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12989-018-0257-1
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