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Propofol induces nuclear localization of Nrf2 under conditions of oxidative stress in cardiac H9c2 cells
Oxidative stress contributes to myocardial ischemia-reperfusion injury, which causes cardiomyocyte death and precipitate life-threatening heart failure. Propofol has been proposed to protect cells or tissues against oxidative stress. However, the mechanisms underlying its beneficial effects are not...
Tallennettuna:
| Julkaisussa: | PLoS One |
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| Päätekijät: | , , , , , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Public Library of Science
2018
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5915683/ https://ncbi.nlm.nih.gov/pubmed/29689082 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0196191 |
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