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Carbon Monoxide Inhibits Islet Apoptosis via Induction of Autophagy

Aim: Carbon monoxide (CO) functions as a therapeutic molecule in various disease models because of its anti-inflammatory and antiapoptotic properties. We investigated the capacity of CO to reduce hypoxia-induced islet cell death and dysfunction in human and mouse models. Results: Culturing islets in...

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Détails bibliographiques
Publié dans:Antioxid Redox Signal
Auteurs principaux: Kim, Do-Sung, Song, Lili, Wang, Jingjing, Wu, Hongju, Gou, Wenyu, Cui, Wanxing, Kim, Jae-Sung, Wang, Hongjun
Format: Artigo
Langue:Inglês
Publié: Mary Ann Liebert, Inc. 2018
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC5905947/
https://ncbi.nlm.nih.gov/pubmed/28826228
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/ars.2016.6979
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