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The beta-adrenergic agonist salbutamol modulates neuromuscular junction formation in zebrafish models of human myasthenic syndromes

Inherited defects of the neuromuscular junction (NMJ) comprise an increasingly diverse range of disorders, termed congenital myasthenic syndromes (CMS). Therapies acting on the sympathetic nervous system, including the selective β2 adrenergic agonist salbutamol and the α and β adrenergic agonist eph...

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Detalhes bibliográficos
Publicado no:Hum Mol Genet
Main Authors: McMacken, Grace, Cox, Dan, Roos, Andreas, Müller, Juliane, Whittaker, Roger, Lochmüller, Hanns
Formato: Artigo
Idioma:Inglês
Publicado em: Oxford University Press 2018
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5905648/
https://ncbi.nlm.nih.gov/pubmed/29462491
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddy062
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