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The PPARγ agonist efatutazone delays invasive progression and induces differentiation of ductal carcinoma in situ
PURPOSE: Ductal carcinoma in situ (DCIS) is a pre-invasive lesion of the breast considered a precursor of invasive ductal carcinoma. This study aimed to determine whether activated PPARγ acts as a tumor suppressor in human DCIS progression. METHODS: We utilized the high-affinity PPARγ agonist, efatu...
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| Foilsithe in: | Breast Cancer Res Treat |
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| Main Authors: | , , , , , , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
2018
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5884724/ https://ncbi.nlm.nih.gov/pubmed/29350308 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10549-017-4649-y |
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